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(242) is that SGLT-1 is expressed mainly in the small intestine, hence its inactivation results in heightened luminal glucose that travels down to the colon where it someway stimulates GLP-1 release
Several pharmacologic and structural properties contribute to this receptor interaction
And that sequestering is like a quarantine and thats what the plaque develops from
The combined effect may contribute to faster improvements in metabolic markers like insulin sensitivity , blood pressure, and lipid profiles
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